sFRP3 and Being sad (a good thing?)

Mice lacking a protein called DKK1 develop more new neurons (right) compared with controls (left).
Mice lacking a protein called DKK1 develop more new neurons (right) compared with controls (left).

Scientist discovered how antidepressant drugs and electroconvulsive therapy works. They found out that these therapy methods reduce the Secreted frizzled-related protein 3 (sFRP3), which is in turn an inhibitor to the creation of new neurons in our brain. (1, 2, 3)

This same protein increases in our body as we get older, thus playing also a role in diseases like Alzheimer. Protein DKK1 also plays a similar role. (4)

Strange. Is it in our Nature to die and get more depressed?

Wow.

If depression is such an integral part of our life, maybe it is a good thing? Could we have overlooked such an important aspect of our existence?

I am leaving now. I am going to go sad…

Author: skakos

Spiros Kakos is a thinker located in Greece. He has been Chief Editor of Harmonia Philosophica since its inception. In the past he has worked as a senior technical advisor for many years. In his free time he develops software solutions and contributes to the open source community. He has also worked as a phD researcher in the Advanced Materials sector related to the PCB industry. He likes reading and writting, not only philosophy but also in general. He believes that science and religion are two sides of the same coin and is profoundly interested in Religion and Science philosophy. His philosophical work is mainly concentrated on an effort to free thinking of "logic" and reconcile all philosophical opinions under the umbrella of the "One" that Parmenides - one of the first thinkers - visualized. The "Harmonia Philosophica" articles program is the tool that will accomplish that. Life's purpose is to be defeated by greater things. And the most important things in life are illogical. We must fight the dogmatic belief in "logic" if we are to stay humans... Credo quia absurdum!

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